cellzscope v2.2.2 software (nanoAnalytics gmbh)
90
Structured Review
nanoAnalytics gmbh
cellzscope v2.2.2 software
Cellzscope V2.2.2 Software, supplied by nanoAnalytics gmbh, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/cellzscope+v2%2E2%2E2+software/cellzscope+software/pmc09910615__pnas__2209944120__sapp-99-4-7
Average 90 stars, based on 1 article reviews
Cellzscope V2.2.2 Software, supplied by nanoAnalytics gmbh, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/cellzscope+v2%2E2%2E2+software/cellzscope+software/pmc09910615__pnas__2209944120__sapp-99-4-7
Average 90 stars, based on 1 article reviews
cellzscope v2.2.2 software - by Bioz Stars,
2026-09
90/100 stars
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other:Article Title: Activation of non-classical NMDA receptors by glycine impairs barrier function of brain endothelial cells Article Snippet: Transendothelial electrical resistance (TEER) measurements were performed using the cellZscope 24-cell module and analyzed with the Article Title: Dietary tryptophan links encephalogenicity of autoreactive T cells with gut microbial ecology. Article Snippet: Transendothelial electrical resistance (TEER) measurements were performed using the cellZscope 24-cell module and Article Title: B7-H1 shapes T-cell–mediated brain endothelial cell dysfunction and regional encephalitogenicity in spontaneous CNS autoimmunity Article Snippet: TEER measurements were performed using the cellZscope 24-cell module and Article Title: Chemokine CCL17 is expressed by dendritic cells in the CNS during experimental autoimmune encephalomyelitis and promotes pathogenesis of disease. Article Snippet: The CC chemokine ligand 17 (CCL17) and its cognate CC chemokine receptor 4 (CCR4) are known to control leukocyte migration, maintenance of TH17 cells, and regulatory T cell (Treg) expansion in vivo.. In this study we characterized the expression and functional role of CCL17 in the pathogenesis of experimental autoimmune encephalomyelitis (EAE).. Using a CCL17/EGFP reporter mouse model, we could show that CCL17 expression in the CNS can be found in a subset of classical dendritic cells (DCs) that immigrate into the CNS during the effector phase of MOG-induced EAE. Article Title: Enhanced pathogenicity of Th17 cells due to natalizumab treatment: Implications for MS disease rebound Article Snippet: Measurement was performed with |